As a further proof of specificity, when cells were previously fixed with p-formaldehyde, permeabilized with 0.
And because phages are large particles, they remain confined to the circulatory system mimicking the conditions to which T.
A screening on the lung-derived endothelial cell line belonging to this panel led to a peptide that homes the lung vasculature in vivo.
Cytokeratins and vimentin are exposed on some endothelial cells Cytokeratins and vimentin are usually recognized as intracellular components of the cytoskeleton, although previous studies have demonstrated the presence of vimentin and cytokeratins on the surface of vascular cells and cardiomyocytes.
The authors have declared that no competing interests exist.
Bladder and other urinary tract disorders have also been described but much less frequently ,.
Significant enrichment of phage particles was also observed with the bladder and esophagus vasculature 213 and 123 times, respectively, relatively to fd-tet , and to a lesser extent, to skeletal muscle endothelium 40 times relative to fd-tet.
Background Transmitted by blood-sucking insects, the unicellular parasite Trypanosoma cruzi is the causative agent of Chagas' disease, a malady manifested in a variety of symptoms from heart disease to digestive and urinary tract dysfunctions.